United States
Department of Transportation
National Highway
Traffic Safety Administration
Paramedic: National Standard Curriculum
(Reprinted with
permission)
http://www.nhtsa.dot.gov/people/injury/ems/
Thoracic Trauma: 7
UNIT TERMINAL
OBJECTIVE
4-7 At
the completion of this unit, the paramedic student will be able to integrate
pathophysiological principles and the assessment findings to formulate a field
impression and implement a treatment plan for a patient with a thoracic injury.
COGNITIVE
OBJECTIVES
At the completion of this unit, the
paramedic student will be able to:
4-7.1 Describe
the incidence, morbidity, and mortality of thoracic injuries in the trauma
patient. (C-1)
4-7.2 Discuss
the anatomy and physiology of the organs and structures related to thoracic
injuries. (C-1)
4-7.3 Predict
thoracic injuries based on mechanism of injury. (C-2)
4-7.4 Discuss
the types of thoracic injuries. (C-1)
4-7.5 Discuss
the pathophysiology of thoracic injuries. (C-1)
4-7.6 Discuss
the assessment findings associated with thoracic injuries. (C-1)
4-7.7 Discuss
the management of thoracic injuries. (C-1)
4-7.8 Identify
the need for rapid intervention and transport of the patient with thoracic
injuries. (C-1)
4-7.9 Discuss
the pathophysiology of specific chest wall injuries, including: (C-1)
a. Rib
fracture
1.
Flail segment
2.
Sternal fracture
4-7.10 Discuss the assessment findings associated with
chest wall injuries. (C-1)
4-7.11 Identify the need for rapid intervention and
transport of the patient with chest wall injuries. (C-1)
4-7.12 Discuss the management of chest wall injuries.
(C-1)
4-7.13 Discuss the pathophysiology of injury to the
lung, including: (C-1)
3.
Simple pneumothorax
4.
Open pneumothorax
5.
Tension pneumothorax
6.
Hemothorax
7.
Hemopneumothorax
8.
Pulmonary contusion
4-7.14 Discuss the assessment findings associated with
lung injuries. (C-1)
4-7.15 Discuss the management of lung injuries. (C-1)
4-7.16 Identify the need for rapid intervention and
transport of the patient with lung injuries. (C-1)
4-7.17 Discuss the pathophysiology of myocardial
injuries, including: (C-1)
a. Pericardial
tamponade
9.
Myocardial contusion
10.
Myocardial rupture
4-7.18 Discuss the assessment findings associated with
myocardial injuries. (C-1)
4-7.19 Discuss the management of myocardial injuries.
(C-1)
4-7.20 Identify the need for rapid intervention and
transport of the patient with myocardial injuries. (C-1)
4-7.21 Discuss the pathophysiology of vascular
injuries, including injuries to: (C-1)
a. Aorta
11.
Vena cava
12.
Pulmonary arteries/
veins
4-7.22 Discuss the assessment findings associated with
vascular injuries. (C-1)
4-7.23 Discuss the management of vascular injuries.
(C-1)
4-7.24 Identify the need for rapid intervention and
transport of the patient with vascular injuries. (C-1)
4-7.25 Discuss the pathophysiology of diaphragmatic
injuries. (C-1)
4-7.26 Discuss the assessment findings associated with
diaphragmatic injuries. (C-1)
4-7.27 Discuss the management of diaphragmatic
injuries. (C-1)
4-7.28 Identify the need for rapid intervention and
transport of the patient with diaphragmatic injuries. (C-1)
4-7.29 Discuss the pathophysiology of esophageal
injuries. (C-1)
4-7.30 Discuss the assessment findings associated with
esophageal injuries. (C-1)
4-7.31 Discuss the management of esophageal injuries.
(C-1)
4-7.32 Identify the need for rapid intervention and
transport of the patient with esophageal injuries. (C-1)
4-7.33 Discuss the pathophysiology of
tracheo-bronchial injuries. (C-1)
4-7.34 Discuss the assessment findings associated with
tracheo-bronchial injuries. (C-1)
4-7.35 Discuss the management of tracheo-bronchial
injuries. (C-1)
4-7.36 Identify the need for rapid intervention and
transport of the patient with tracheo-bronchial injuries. (C-1)
4-7.37 Discuss the pathophysiology of traumatic
asphyxia. (C-1)
4-7.38 Discuss the assessment findings associated with
traumatic asphyxia. (C-1)
4-7.39 Discuss the management of traumatic asphyxia.
(C-1)
4-7.40 Identify the need for rapid intervention and
transport of the patient with traumatic asphyxia. (C-1)
4-7.41 Integrate the pathophysiological principles to
the assessment of a patient with thoracic injury. (C-1)
4-7.42 Differentiate between thoracic injuries based
on the assessment and history. (C-3)
4-7.43 Formulate a field impression based on the
assessment findings. (C-3)
4-7.44 Develop a patient management plan based on the
field impression. (C-3)
AFFECTIVE OBJECTIVES
At the completion of this unit, the paramedic student will be able to:
4-7.45 Advocate the use of a thorough assessment to
determine a differential diagnosis and treatment plan for thoracic trauma.
(A-3)
4-7.46 Advocate the use of a thorough scene survey to
determine the forces involved in thoracic trauma. (A-3)
4-7.47 Value the implications of failing to properly
diagnose thoracic trauma. (A-2)
4-7.48 Value the implications of failing to initiate
timely interventions to patients with thoracic trauma. (A-2)
PSYCHOMOTOR OBJECTIVES
At the completion of this unit, the paramedic student will be able to:
4-7.49 Demonstrate a clinical assessment for a patient
with suspected thoracic trauma. (P-1)
4-7.50 Demonstrate the following techniques of
management for thoracic injuries: (P-1)
a. Needle decompression
13.
Fracture stabilization
14.
Elective intubation
15.
ECG monitoring
16.
Oxygenation and
ventilation
DECLARATIVE
I. Introduction
A. Epidemiology
1. Incidence
2. Morbidity and mortality of thoracic
injuries
3. Risk factors
4. Prevention strategies
a. Gun safety education
b. Sports training
c. Seat belts
d. Other
B. Mechanism of injury
1. Classification
a. Blunt thoracic injuries
(1) Deceleration
(2) Compression
b. Penetrating thoracic injuries
2. Injury patterns
a. General Types
(1) Open injuries
(2) Closed Injuries
b. Thoracic cage
c. Cardiovascular
d. Pleural and pulmonary
e. Mediastinal
f. Diaphragmatic
g. Esophageal
h. Penetrating cardiac trauma
3. Blast injury
a. Confined spaces
b. Shock wave
C. Anatomy and physiology review of the
thorax
1. Anatomy
a. Skin
b. Bones
(1) Thoracic cage
(2) Sternum
(3) Thoracic spine
c. Muscles
(1) Intercostal
(2) Trapezius
(3) Latisissimus dorsi
(4) Rhomboids
(5) Pectoralis major
(6) Diaphragm
(7) Sternocleidomastoid
d. Trachea
e. Bronchi
f. Lungs
(1) Parenchyma
(2) Alveoli
(3) Alveolar - capillary interface
(4) Pleura
(a) Visceral
(b) Parietal
(c) Serous fluid
(5) Lobes
g. Vessels
(1) Arteries
(a) Aorta
(b) Carotid
(c) Subclavian
(d) Intercostal arteries
(e) Innominate
(f) Internal mammary
(2) Veins
(a) Superior vena cava
(b) Inferior vena cava
(c) Subclavian
(d) Internal jugular
(3) Pulmonary
(a) Arteries
(b) Veins
h. Heart
(1) Ventricles
(2) Atria
(3) Valves
(4) Pericardium
i. Esophagus
(1) Thoracic inlet
(2) Course through chest
(3) Esophageal foramen through diaphragm
j. Mediastinum
(1) Structures located in mediastinum
(a) Heart
(b) Trachea
(c) Vena cava
(d) Aorta
(e) Esophagus
2. Physiology
a. Ventilation
(1) Expansion and contraction of thoracic
cage
(a) Bellows system
(b) Musculoskeletal structure
(c) Intercostal muscles
(d) Diaphragm
(e) Accessory muscles
(f) Changes in intrathoracic pressure
b. Respiration
(1) Neurochemical control
(2) Gas exchange
(a) Alveolar-capillary interface
(b) Capillary-cellular interface
(c) Pulmonary circulation
(d) Cardiac circulation
(e) Acid-base balance
i) Henderson-Hasselbach equation
ii) Respiratory alkalosis
iii) Respiratory acidosis
iv) Compensation for metabolic acidosis and
alkalosis
II. General system pathophysiology,
assessment and management of thoracic trauma
A. Pathophysiology
1. Impairments in cardiac output
a. Blood loss
b. Increased intrapleural pressures
c. Blood in pericardial sac
d. Myocardial valve damage
e. Vascular disruption
2. Impairments in ventilatory efficiency
a. Chest bellow action compromise
(1) Pain restricting chest excursion
(2) Air entering pleural space
(3) Chest wall fails to move in unison
b. Bleeding in pleural space
c. Ineffective diaphragmatic contraction
3. Impairments in gas exchange
a. Atelectasis
b. Contused lung tissue
c. Disruption of respiratory tract
B. Assessment findings
1. Pulse
a. Deficit
b. Tachycardia
c. Bradycardia
2. Blood pressure
a. Narrow pulse pressure
b. Hypertension
c. Hypotension
d. Pulsus paradoxus
3. Respiratory rate and effort
a. Tachypnea
b. Bradypnea
c. Labored
d. Retractions
e. Other evidence of respiratory distress
4. Possible hypothermia
5. Skin
a. Diaphoresis
b. Pallor
c. Cyanosis
d. Open wounds
e. Ecchymosis
f. Other evidence of trauma
6. Hemoptysis
7. Neck
a. Position of trachea
b. Subcutaneous emphysema
c. Jugular venous distention
d. Penetrating wounds
8. Chest
a. Contusions
b. Tenderness
c. Asymmetry
d. Lung sounds
(1) Absent or decreased
(a) Unilateral
(b) Bilateral
(2) Location
(3) Bowel sounds in hemithorax
e. Abnormal percussion finding
(1) Hyperresonance
(2) Hyporesonance
f. Heart sounds
(1) Muffled
(2) Distant
(3) Regurgitant murmur
g. Shift of apical impulse
h0 Open wounds
i0 Impaled object or penetration
j0 Crepitation
k0 Paradoxical movement of chest wall segment
9 Scaphoid abdomen
10 Decreased level of consciousness
11 ECG
a0 ST - T wave elevation or depression
b0 Conduction disturbances
c0 Rhythm disturbances
12 History
a0 Dyspnea
b0 Chest pain
c0 Associated symptoms
(1) Other areas of pain or discomfort
(2) Symptoms prior to incident
d0 Past history of cardiorespiratory
disease
e0 Use of restraint in motor vehicle crash
C0 Management
1 Airway
and ventilation
a0 Oxygen therapy
b0 Endotracheal intubation
c0 Needle cricothyrotomy
d0 Surgical cricothyrotomy
e0 Positive pressure ventilation
f0 Occlude open wounds
g0 Stabilize chest wall
2 Circulation
a0 Manage cardiac dysrhythmias
b0 Intravenous access
3 Pharmacologic
a0 Analgesics
b0 Antiarrhythmics
4 Non-pharmacologic
a0 Needle thoracostomy
b0 Tube thoracostomy - in hospital management
c0 Pericardiocentesis - in hospital management
5 Transport considerations
a0 Appropriate mode
b0 Appropriate facility
III Chest wall injuries
A0 Rib fractures
1 Epidemiology
a0 Incidence
(1) Infrequent until adult life
(2) Most often elderly patients
(3) Significant force required
b0 Morbidity/ mortality
(1) Can lead to serious consequences
(2) Older ribs more brittle and rigid
(3) Associated underlying pulmonary or
cardiovascular injury
(4) Increases with
(a) Age
(b) Number of fractures
(c) Location of fractures
2 Anatomy and physiology review
3 Pathophysiology
a0 Most often caused by blunt trauma,
bowing effect with midshaft fracture
b0 Ribs 4 to 9 are most often fractured (thin and poorly
protected)
c0 Respiratory restriction due to pain and splinting
(1) Atelectasis
(2) Ventilation/ perfusion mismatch
d0 May be associated with underlying lung
or cardiac contusion
e0 Intercostal vessel injury
f0 Associated complications
(1) First and second ribs are injured by
severe trauma
(a) Rupture of aorta
(b) Tracheobronchial tree injury
(c) Vascular injury
(2) Left lower rib injury associated with
splenic rupture
(3) Right lower rib injury associated with
hepatic injury
(4) Multiple rib fractures
(a) Atelectasis
(b) Hypoventilation
(c) Inadequate cough
(d) Pneumonia
(5) Open rib fracture associated with
visceral injury
(6) Posterior rib fracture
(a) Fifth through ninth ribs most frequently
injured
(b) Lower ribs associated with spleen and
kidney injury
4 Assessment findings
a0 Localized pain
b0 Pain that worsens
(1) Movement
(2) Deep breathing
(3) Coughing
c0 Point tenderness
d0 Crepitus or audible crunch
e0 Splinting on respiration
f0 Anteroposterior pressure elicits pain
5 Management
a0 Airway and ventilation
(1) Oxygen therapy
(2) Positive pressure ventilation
(3) Encourage coughing and deep breathing
b0 Pharmacological
(1) Analgesics
c0 Non-pharmacological
(1) Splint - but avoid circumferential
splinting
d0 Transport consideration
(1) Appropriate mode
(2) Appropriate facility
e0 Psychological support/ communication
strategies
B0 Flail segment
1 Epidemiology
a0 Incidence
(1) Most common cause is vehicular crash
(2) Falls from heights
(3) Industrial accidents
(4) Assault
(5) Birth trauma
b0 Morbidity/ mortality
(1) Significant chest trauma
(2) Mortality rates 20-40% due to associated
injuries
(3) Mortality increased with
(a) Advanced age
(b) Seven or more rib fractures
(c) Three or more associated injuries
(d) Shock
(e) Head injuries
2 Pathophysiology
a0 Three or more ribs fractured in two or
more places producing a free floating segment of chest wall
b0 Respiratory failure due to
(1) Underlying pulmonary contusion
(2) Associated intrathoracic injury
(3) Inadequate bellow action of chest
c0 Paradoxical movement of the chest
(1) Minimal because of muscle spasm
(2) Must be large to compromise ventilation
d0 Pain
(1) Reduces thoracic expansion
(2) Decreases ventilation
e0 Pulmonary contusion
(1) Decreased lung compliance
(2) Intra alveolar-capillary hemorrhage
(3) Alveolar hemorrhage
f0 Decreased ventilation
g0 Impaired venous return with resultant ventilation-perfusion
mismatch
h0 Hypercapnia
i0 Hypoxia
3 Assessment findings
a0 Chest wall contusion
b0 Respiratory distress
c0 Paradoxical chest wall movement
d0 Pleuritic chest pain
e0 Crepitus
f0 Pain and splinting of affected side
g0 Tachypnea
h0 Tachycardia
i0 Possible bundle branch block on ECG
4 Management
a0 Airway and ventilation
(1) Positive pressure ventilation may be
needed
(2) Oxygen (high concentration)
(3) Evaluate the need for endotracheal
intubation
(4) Stabilize flail segment (may be
controversial locally)
(5) Positive end expiratory pressure (PEEP)
b0 Circulation
(1) Restrict fluids
c0 Pharmacologic
(1) Analgesics
d0 Non-pharmacologic
(1) Positioning
(2) Endotracheal intubation and positive
pressure ventilation for internal splinting effect
e0 Transport considerations
(1) Appropriate mode
(2) Appropriate facility
f0 Psychological support/ communication
strategies
C0 Sternal fracture
1 Epidemiology
a0 Incidence
(1) 5-8% in blunt chest trauma
(2) Deceleration compression injury
(a) Steering wheel
(b) Dashboard
(3) Blow to chest
(4) Severe hyperflexion of thoracic cage
(5) Occur at or below the manubriosternal
junction
b0 Morbidity/ mortality
(1) 25-45% mortality
(2) High association with myocardial or lung
injury
(a) Myocardial contusion
(b) Myocardial rupture
(c) Pulmonary contusion
2 Pathophysiology
a0 Associated injuries cause morbidity and
mortality
(1) Pulmonary and myocardial contusion
(2) Flail chest
(3) Vascular disruption of thoracic vessels
(4) Intraabdominal injuries
(5) Head injuries
b0 Rarely is fracture displaced
posteriorly to directly impinge on heart or vessels
3 Assessment findings
a0 Localized pain
b0 Tenderness over sternum
c0 Crepitus
d0 Tachypnea
e0 ECG changes associated with myocardial contusion
f0 History of blunt trauma
4 Management
a0 Airway and ventilation
b0 Circulation
(1) Restrict fluids if pulmonary contusion
is suspected
5 Pharmacologic
a0 Analgesics
6 Non-pharmacologic
a0 Allow chest wall self-splinting
7 Transport considerations
a0 Appropriate mode
b0 Appropriate facility
8 Psychological support/ communication
strategies
IV Injury
to the lung
A0 Simple pneumothorax
1 Epidemiology
a0 Incidence
(1) 10-30% in blunt chest trauma
(2) Almost 100% with penetrating chest
trauma
b0 Morbidity/ mortality
(1) Extent of atelectasis
(2) Associated injuries
2 Pathophysiology
a0 Lung 1-3 cm away from the chest wall
b0 May have stable amount of accumulation of air
c0 Pulmonary function may be good
d0 Internal wound allows air to enter the pleural space
e0 Small tears self-seal, larger one may progress
f0 Paper bag syndrome
g0 If standing air will accumulate in the apices, check there
first for diminished breath sounds otherwise, if supine it accumulates in the
anterior chest
h0 Trachea may tug towards the effected side
i0 Ventilation/ perfusion mismatch
3 Assessment findings
a0 Tachypnea
b0 Tachycardia
c0 Respiratory distress
d0 Absent or decreased breath sounds on affected side
e0 Hyperresonance
f0 Decreased chest wall movement
g0 Dyspnea
h0 Chest pain referred to shoulder or arm on affected side
i0 Slight pleuritic chest pain
4 Management
a0 Airway and ventilation
(1) Positive pressure ventilation if
necessary
(2) Monitor for
development of tension pneumothorax
b0 Non-pharmacologic
(1) Needle thorocostomy
c0 Transport consideration
(1) Appropriate mode
(2) Appropriate facility
5 Psychological support/ communication
strategies
B0 Open pneumothorax
1 Epidemiology
a0 Incidence
(1) Penetrating trauma
b0 Morbidity/ mortality
(1) Profound hypoventilation could result
(2) Death related to delayed management
2 Pathophysiology
a0 Open defect in the chest wall
(1) Allows communication between pleural
space and atmosphere
(2) Prevents development of negative
intrapleural pressure
(3) Produces collapse of ipsilateral lung
(4) Inability to ventilate affected lung
(5) Ventilation/ perfusion mismatch
(a) Shunting
(b) Hypoventilation
(c) Hypoxia
(d) Large functional dead space
b0 Air will enter pleural space during
inspiratory phase
c0 Air may exit during exhalation phase
d0 Resistance to air flow through respiratory tract may be greater
than through open wound resulting in ineffective respiratory effort
e0 One way flap valve may let air in but not out resulting in
built up pressure in pleural space
f0 Direct lung injury may be present
g0 Vena cava kinked from swaying of mediastinum
h0 Preload decreased from knifing of inferior vena cava
3 Assessment findings
a0 To and fro air motion out of defect
b0 Defect in the chest wall
c0 Penetrating injury to the chest which does not seal itself
d0 Sucking sound on inhalation
e0 Tachycardia
f0 Tachypnea
g0 Respiratory distress
h0 Subcutaneous emphysema
i0 Decreases breath sounds on affected side
4 Management
a0 Airway and ventilation
(1) Positive pressure ventilation if
necessary
(2) Monitor for development of tension
pneumothorax
b0 Non-pharmacologic
(1) Occlude open wound
(2) Tube thoracostomy - in hospital management
c0 Transport consideration
(1) Appropriate mode
(2) Appropriate facility
5 Psychological support/ communication
strategies
C0 Tension pneumothorax
1 Epidemiology
a0 Incidence
(1) Penetrating trauma
(2) Blunt trauma
b0 Morbidity/ mortality
(1) Profound hypoventilation could result
(2) Death related to delayed management
(3) Immediate life-threatening chest injury
2 Pathophysiology
a0 Defect in airway allowing communication
with pleural space
b0 Blunt trauma
(1) Penetration by rib fracture
(2) Sudden increase in intrapulmonary
pressure
(3) Bronchial disruption from shear forces
c0 Air trapped in pleural space with build
up of pressure
d0 Lung collapse on affected side with mediastinal shift to
contralateral side
e0 Lung collapse leads to right-to-left intrapulmonary shunting
and hypoxia
f0 Reduction in cardiac output
(1) Increased intrathoracic pressure
(2) Deformation of vena cava reducing
preload (decreased venous return to heart)
3 Assessment findings
a0 Unilateral decreased or absent breath
sounds
b0 Dyspnea
c0 Tachypnea
d0 Respiratory distress
e0 Extreme anxiety
f0 Cyanosis
g0 Bulging of intercostal muscles
h0 Tachycardia
i0 Hypotension
j0 Narrow pulse pressure
k0 Subcutaneous emphysema
l0 Jugular venous distention
m0 Tracheal deviation
n0 Hyperresonance
4 Management
a0 Airway and ventilation
(1) Positive pressure ventilation if
necessary
b0 Circulation
(1) Relieve tension pneumothorax to improve
cardiac output
c0 Non-pharmacologic
(1) Occlude open wound
(2) Needle thoracentesis
(a) Equipment
(b) Technique
(c) Assess the need for a second or third
needle insertion
(3) Tube thoracostomy - in hospital management
d0 Transport consideration
(1) Appropriate mode
(2) Appropriate facility
e0 Psychological support/ communication
strategies
D0 Hemothorax
1 Epidemiology
a0 Incidence
(1) Associated with pneumothorax
(2) Blunt or penetrating trauma
(3) Rib fractures are frequent cause
b0 Morbidity/ mortality
(1) Life-threatening injury that frequently
requires urgent chest tube and/ or surgery
(2) Hemothorax associated with great vessel
or cardiac injury
(a) 50% will die immediately
(b) 25% live five to ten minutes
(c) 25% may live 30 minutes or longer
2 Pathophysiology
a0 Accumulation of blood in the pleural
space
b0 Bleeding from
(1) Penetrating or blunt lung injury
(2) Chest wall vessels
(3) Intercostal vessels
(4) Myocardium
c0 Pulmonary parenchyma is low-pressure
vascular system
d0 Bleeding from pulmonary contusion generally causes 1000 to
1500 cc blood loss
e0 Massive hemothorax indicates great vessel or cardiac injury
f0 Collapse of ipsilateral lung
g0 Respiratory insufficiency dependent on amount of blood
h0 Hypoxia
i0 Hypotension and inadequate perfusion may result from blood
loss
j0 Chest cavity can hold 2,000 to 3,000 ml of blood
k0 Classified by amount of blood loss
l. Tissue pressure effects of legs, arms
and abdomen versus thorax
(1 La Place law
(2 Extraluminal pressure in legs
(3 Extraluminal pressure in thorax
m. An intercostal artery can easily bleed
50 ccs per minute
n. Intrapulmonary hemorrhage
(1 Bronchus
(2 Parenchyma
3. Assessment findings
a. Tachypnea
b. Tachycardia
c. Dyspnea
d. Respiratory distress
e. Hypotension
f. Narrow pulse pressure
g. Pleuritic chest pain
h. Pale, cool, moist skin
i. Dullness on percussion
j. Decreased breath sounds
4. Management
a. Airway and ventilation
(1 Positive pressure ventilation if
necessary
b. Circulation
(1 Re-expand the affected lung to reduce
bleeding
c. Non-pharmacological
(1 Needle chest decompression
(2 Tube thoracostomy - in hospital
management
d. Transport considerations
(1 Appropriate mode
(2 Appropriate facility
e. Psychological support/ communication
strategies
E. Hemopneumothorax
1. Pathophysiology
a. Pneumothorax with bleeding in pleural
space
2. Assessment
a. Findings and management same as
hemothorax
3. Management
a. Management is the same as a hemothorax
F. Pulmonary contusion
1. Epidemiology
a. Incidence
(1 Blunt trauma to chest
(a Most common injury from blunt thoracic
trauma
(b 30-75% with blunt trauma have pulmonary contusion
(2 Associated commonly with rib fracture
(3 High energy shock waves from explosion
(4 High velocity missile wounds
(5 Rapid deceleration
(6 High incidence of extrathoracic injuries
(7 Low velocity - ice pick
b. Morbidity/ mortality
(1 Missed due to high incidence of other
associated injuries
(2 Mortality between 14-20%
2. Pathophysiology
a. Three physical mechanisms
(1 Implosion effect
(a Overexpansion of air in lungs secondary to positive-pressure
concussive wave
(b Rapid excessive stretching and tearing of alveoli
(2 Inertial effect
(a Strips alveoli from heavier bronchial structures when
accelerated at varying rates by concussive wave
(3 Spalding effect
(a Liquid-gas interface is disrupted by shock-wave
(b Wave releases energy
(c Differential transmission of energy causes disruption of
tissue
b. Alveolar and capillary damage with
interstitial and intraalveolar extravasation of blood
c. Interstitial edema
d. Increased capillary membrane
permeability
e. Gas exchange disturbances
f. Hypoxemia and carbon dioxide retention
g. Hypoxia causes reflex thickening of
mucous secretions
(1 Bronchiolar obstruction
(2 Atelectasis
h. Blood is shunted away from unventilated
alveoli leading to further hypoxemia
3. Assessment findings
a. Tachypnea
b. Tachycardia
c. Cough
d. Hemoptysis
e. Apprehension
f. Respiratory distress
g. Dyspnea
h. Evidence of blunt chest trauma
i. Cyanosis
4. Management
a. Airway and ventilation
(1 Positive pressure ventilation if necessary
b. Circulation
(1 Restrict intravenous fluids (use caution
restricting fluids in hypovolemic patients)
c. Transport considerations
(1 Appropriate mode
(2 Appropriate facility
d. Psychological support/ communication
strategies
V. Myocardial injuries
A. Pericardial tamponade
1. Epidemiology
a. Incidence
(1 Rare in blunt trauma
(2 Penetrating trauma
(3 Occurs in less than 2% of chest trauma
b. Morbidity/ mortality
(1 Gunshot wounds carry higher mortality
than stab wounds
(2 Lower mortality rate if isolated
tamponade is present
2. Anatomy and physiology
a. Pericardium
(1 Tough fibrous sac
(2 Encloses heart
(3 Attaches to great vessels at the base of
heart
(4 Two layers
(a Visceral forms epicardium
(b Parietal regarded as sac itself
(5 Purposes
(a Anchor heart
(b Restricts excess movement
(c Prevents kinking of great vessels
(6 Parietal layer is acutely nondispensable
but can chronically distend by as much as 1,000 to 1,500 ml
(7 Space between visceral and parietal
layer is "potential space"
(8 Space normally filled with 30-50 ml of
straw-colored fluid secreted by visceral layer
(a Lubrication
(b Lymphatic drainage
(c Immunologic protection for heart
3. Pathophysiology
a. Rapid accumulation of fluid over a
period of minutes to hours leads to increases in intrapericardial pressure
b. Increased intrapericardial pressure
(1 Compresses heart and decreases cardiac
output due to restricted diastolic expansion and filling
(2 Hampers venous return
c. Myocardial perfusion decreases due to
pressure effects on walls of heart and decreased diastolic pressures
d. Ischemic dysfunction may result in
infarction
e. Removal of as little as 20 ml of blood
may drastically improve cardiac output
4. Assessment findings
a. Tachycardia
b. Respiratory distress
c. Narrow pulse pressure
d. Pulsus paradoxus
e. Cyanosis
(1 Head
(2 Neck
(3 Upper extremities
f. Beck’s triad - advanced stage seen in
only 30% of patients
(1 Hypotension
(2 Neck vein distention
(3 Muffled heart tones
g. Kussmaul's sign
h. ECG changes
5. Management
a. Airway and ventilation
b. Circulation
(1 Fluid challenge
c. Non-pharmacological
(1 Pericardiocentesis - in hospital
management
d. Transport considerations
(1 Appropriate mode
(2 Appropriate facility
e. Psychological support/ communication
strategies
B. Myocardial contusion (blunt myocardial
injury)
1. Epidemiology
a. Incidence
(1 16-76% of blunt trauma
b. Morbidity/ mortality
(1 Significant cause of morbidity and
mortality in the blunt trauma patient
2. Pathophysiology
a. Hemorrhage with edema and fragmented
myocardial fibers
b. Cellular injury
c. Vascular damage may occur
d. Hemopericardium may occur from
lacerated epicardium or endocardium
e. Fibrinous reaction at contusion site
may lead to
(1 Delayed
rupture
(2 Ventricular aneurysm
f. Areas of damage are well demarcated
g. Conduction defects
3. Assessment findings
a. Associated injuries
(1 One to three rib fractures
(2 Sternal fracture
b. Retrosternal chest pain
c. ECG changes
(1 Persistent tachycardia
(2 ST elevation, T wave inversion
(3 Right bundle branch block
(4 Atrial flutter, fibrillation
(5 Premature ventricular contractions
(6 Premature atrial contractions
d. New cardiac murmur
e. Pericardial friction rub (late)
4. Management
a. Airway and ventilation
(1 Oxygen therapy
b. Circulation
(1 Intravenous fluid volume
c. Pharmacological
(1 Antiarrhythmics
(2 Vasopressors
d. Transport considerations
(1 Appropriate mode
(2 Appropriate facility
e. Psychological support/ communication
strategies
C. Myocardial rupture
1. Associated with immediate trauma or
delayed for 2-3 weeks
2. Associates with blunt trauma
a. Compression between sternum and vertebrae
3. Penetrating trauma
a. Rib
b. Missile
c. Sternal bone
4. History of trauma with a presentation
of
a. Congestive heart failure
b. Cardiac tamponade
5. Immediate onset of congestive heart
failure following trauma
a. Rupture of cardiac valves
b. Intraventricular septal rupture
6. Management is supportive
VI. Vascular injuries
A. Aortic dissection/ rupture
1. Epidemiology
a. Incidence
(1 Blunt trauma
(a Motor vehicle crash
(b Falls
(2 15% of all blunt trauma deaths
2. Morbidity/ mortality
a. 85-95% die instantaneously
b. 10-15% survive to arrive at hospital
(1 33% of survivors die within six hours
(2 33% of survivors die within twenty-four
hours
(3 33% survive three days or longer
3. Pathophysiology
a. Shear injury
b. Separation of the aortic intima and
media
c. Blood enters media through a small
intima tear
d. Tear due to effect of high speed
deceleration on portions of the aorta at points of relative fixation
e. Increased intraluminal pressure results
from impact
f. Thinned out layer may rupture
g. Descending aorta at the isthmus just
distal to left subclavian artery is most common site of rupture (ligamentum
arteriosom)
h. Ruptures of ascending aorta much less
common
4. Assessment findings
a. Retrosternal or interscapular pain
b. Dyspnea
c. Dysphagia
d. Ischemic pain of the extremities
e. Upper extremity hypertension with
absent or decreased amplitude of femoral pulses
f. Harsh systolic murmur over precordium
or interscapular region
5. Management
a. Airway and ventilation
b. Circulation
(1 Do not over hydrate
c. Transport considerations
(1 Appropriate mode
(2 Appropriate facility
d. Psychological support/ communication
strategies
B. Penetrating wounds of the great vessels
1. Usually involve
a. Chest
b. Abdomen
c. Neck
2. Wounds are accompanied by
a. Massive hemothorax
b. Hypovolemic shock
c. Cardiac tamponade
d. Enlarging hematomas
3. Hematomas may cause compression of any
structure
a. Vena cava
b. Trachea
c. Esophagus
d. Great vessels
e. Heart
4. Management
a. Manage hypovolemia
(1 PASG not recommended
b. Relief of tamponade if present
c. Expeditious transport
VII. Other thorax injuries
A. Diaphragmatic injury
1. Epidemiology
a. Incidence
(1 Blunt trauma
(2 Penetrating trauma
(3 Frequently encountered injury
b. Morbidity/ mortality
(1 Could be life-threatening
2. Pathophysiology
a. High-pressure compression to abdomen
with resultant intra-abdominal pressure increase
b. Can produce very subtle signs and
symptoms
c. Bowel obstruction and strangulation
d. Restriction of lung expansion
(1 Hypoventilation
(2 Hypoxia
e. Mediastinal shift
(1 Cardiac compromise
(2 Respiratory compromise
3. Assessment findings
a. Tachypnea
b. Tachycardia
c. Respiratory distress
d. Dullness to percussion
e. Scaphoid abdomen
f. Bowel sounds in affected hemithorax
g. Decreased breath sounds
4. Management
a. Airway and ventilation
(1 Positive pressure ventilation if
necessary
(2 Caution IPPB may worsen the injury
b. Non-pharmacologic
(1 Do not place patient in Trendelenburg position
c. Transport consideration
(1 Appropriate
mode
(2 Appropriate facility
d. Psychological support/ communication
strategies
B. Esophageal injury
1. Epidemiology
a. Incidence
(1 Penetrating trauma most frequent cause
(2 Rare in blunt trauma
b. Morbidity/ mortality
(1 Could be life-threatening if missed
2. Pathophysiology
a. Missile and knife wounds penetrate
esophagus
b. Can perforate spontaneously
(1 Violent emesis
(2 Carcinoma
(3 Anatomic distortions produced by
diverticulae or gastric reflux
3. Assessment findings
a. Pain
b. Fever
c. Hoarseness
d. Dysphagia
e. Respiratory distress
f. Cervical esophageal perforation
(1 Local tenderness
(2 Subcutaneous emphysema
(3 Resistance of neck on passive motion
g. Intrathoracic esophageal perforation
(1 Mediastinal emphysema
(2 Mediastinitis
(3 Subcutaneous emphysema
(4 Mediastinal crunch
(5 Splinting of chest wall
h. Respiratory distress
i. Shock
4. Management
a. Airway and ventilation
b. Transport consideration
(1 Appropriate mode
(2 Appropriate facility
c. Psychological support/ communication
strategies
C. Tracheo-bronchial injuries
1. Epidemiology
a. Incidence
(1 Rare injury - less than 3% of chest
trauma
(2 Penetrating trauma
(3 Blunt trauma
b. Morbidity/ mortality
(1 High mortality rate - greater than 30%
2. Pathophysiology
a. Majority occur within 3 cm of carina
b. Tear can occur anywhere along tracheal/
bronchial tree
c. Rapid movement of air into pleural
space
d. Tension pneumothorax refractory to
needle decompression
e. Continuous flow of air from needle of
decompressed chest
f. Severe hypoxia
3. Assessment
a. Tachypnea
b. Tachycardia
c. Massive subcutaneous emphysema
d. Dyspnea
e. Respiratory distress
f. Hemoptysis
g. Signs of tension pneumothorax that
doesn't respond to needle decompression
4. Management
a. Airway and ventilation
b. Circulation
c. Transport consideration
(1 Appropriate mode
(2 Appropriate facility
D. Traumatic asphyxia
1. Epidemiology
a. Incidence
b. Morbidity/ mortality
2. Pathophysiology
a. Sudden compressional force squeezes the
chest
b. Blood backs up into the head and neck
c. Jugular veins engorge, capillaries
rupture
3. Assessment
a. Cyanosis to the face and upper neck
b. Jugular venous distention
c. Swelling or hemorrhage of the
conjunctiva
d. Skin below area remains pink
e. Hypotension when pressure released
4. Management
a. Airway and ventilation
b. Circulation
(1) Expect hypotension once compression is
released
c. Pharmacological
(1) Sodium bicarbonate should be guided by
ABGs in hospital
d. Transport considerations
(1) Appropriate mode
(2) Appropriate facility
VIII.
Integration